The duration and magnitude of hyperuricaemia is directly correlated with the likelihood of developing gouty arthritis and developing uric acid kidney stones. The relationship between MRI appearance and clinically relevant intraoperative findings is incompletely understood. The increased risk of cardiovascular morbidity and mortality in rheumatoid arthritis and gout has been increasingly acknowledged in past decades, with accumulating evidence that gout, just as with rheumatoid arthritis, is an independent cardiovascular risk factor. Noninfectious inflammatory tenosynovitis of the digital flexor tendons and other tendons of the hand may be due to crystalline deposition (eg, gout or calcium pyrophosphate disease [CPPD; pseudogout]), inflammatory arthropathy (eg, rheumatoid arthritis [RA]), or reactions to penetrating wounds from sharp objects (eg, rose thorns or sea urchin spines). Buildup of uric acid crystals in a joint causes gouty arthritis. Gout can be defined as arthritis due to deposition of monosodium urate (MSU) crystals within joints causing acute inflammation and eventual tissue damage [].. 1–3 Some studies have reported that the prevalence of gout is from 1% to 3% in Europe and 3.9% in the US. 1. Symptoms and signs include joint pain, swelling, heat, and redness, typically of a single joint. 4, 5 Also, more than 7.44 million cases of gout were estimated to have occurred worldwide in 2017. The goal of this study was to evaluate capitellar OCD lesions via preoperative MRI and to determine its accuracy in predicting lesion characteristics including lesion size and capitellar lateral wall integrity. Hyperuricemia is one of the major metabolic diseases associated with gout which is on a rise in many countries. Introduction. Gout may be treated with diet and lifestyle changes, as well as medication.
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